Is Green Tea Good For The Liver?
Green tea is an antioxidant-rich beverage consumed by millions of people around the world. It has a number of well-established health benefits, but is it good for your liver?
Tea is an infusion of the leaves of the Camellia sinensis plant. Green, white, oolong and black tea are all produced from the leaves of the same plant. Different processing methods yield the various types of tea. All tea contains some flavonoids, caffeine, fluoride and theanine. Green tea contains the antioxidant catechins called EGCG and ECG. Studies have shown these antioxidants to have a range of health benefits.
Some of these include:
- Reduced risk of liver cancer
- Promoting healthy gut bugs by acting as a prebiotic
- Reducing inflammation associated with autoimmune disease
- A reduction in anxiety
Most studies used a daily dose of 4 cups of green tea.
You may have read articles on the internet purporting that green tea is harmful to the liver. I do not agree with this claim. There have been some isolated cases of individuals suffering liver damage that have been tied to green tea, however an exact cause of the liver damage has never been identified.
The incidents involved green tea as a component in a weight loss supplement. Many weight loss formulas contain banned ingredients designed to rev up the body’s metabolism. Quality control is often very poor for weight loss supplements, and they sometimes contain poor quality, contaminated ingredients. They are often manufactured in countries with poor quality control standards.
I have included green tea in Livatone Plus capsules specifically because it reduces inflammation and protects the liver. Livatone Plus is manufactured in a GMP facility in the USA from the highest quality and purest raw materials.
Anil K. Singh, Sadiq Umar, Sharayah Riegsecker, Mukesh Chourasia, Salahuddin Ahmed. Regulation of Transforming Growth Factor β-Activated Kinase Activation by Epigallocatechin-3-Gallate in Rheumatoid Arthritis Synovial Fibroblasts: Suppression of K63-Linked Autoubiquitination of Tumor Necrosis Factor Receptor-Associated Factor 6. Arthritis & Rheumatology, 2016; 68 (2): 347